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PBRT, and STAD): Beginning 2 months before the start of radiotherapy, patients receive STAD therapy as in arm II.

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LHRH agonists are decapeptides that exert a nonpulsatile, constant stimulation to the anterior pituitary gland, which in turn decreases LH and testosterone production.

In the 1990s, the advent of intensity modulation radiotherapy (IMRT) and image guidance radiotherapy either with transabdominal ultrasound or the intraprostatic placement of fiducial markers further refined treatment delivery.

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In a multicenter, double-blind, controlled clinical trial, 813 patients with previously untreated advanced prostate cancer were to receive CASODEX (bicalutamide) 50 mg once daily (404 patients) or flutamide 250 mg (409 patients) three times a day, each in combination with LHRH analogs (either goserelin acetate implant or leuprolide acetate depot).

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For the subset of these who develop metastatic disease, the opportunity to receive minimally toxic, oral, endocrine therapy with clinical benefit is a new treatment option.

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Initial treatment with diethylstilbestrol ( DES) was followed by maximal androgen blockade ( MAB) therapy using bicalutamide.

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ABSTRACT: For patients with advanced prostate cancer, luteinizing hormone-releasing hormone (LHRH) agonists have provided successful androgen deprivation therapy (ADT) for some 25 years.

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It was recognized in the 1940s that 17-alpha alkyl substitution retards the presystemic metabolism of testosterone, extending its half-life and making it orally active.

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