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Aims: Therapeutic drug monitoring of tacrolimus in post transplant patients has been deemed to be extremely critical due to individual variability in tacrolimus pharmacokinetics (PK), which was attributed to various demographic, hematological, time-dependent (posttransplant), genetic, and ethnic factors.
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National Cancer Institutedesignated cancer centers and randomly surveyed 71 other cancer centers.
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Patients experiencing post-transplant hepatic impairment may be associated with an increased risk of developing renal insufficiency related to high blood levels of this drug.
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Although the prognosis of SRNS is complicated and unfavorable, intensive treatment in the early stage of the disease may achieve remission in more than half of the patients.
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Toxicity to this illicit substance continues to be the leading cause of referral to the poison center.
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The differences that we observed in mean tacrolimus trough levels before and after conversion did not result in clinically relevant differences in graft function.
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The present study aims to develop a population pharmacokinetic (POP-PK) model to define the PK in pediatric renal transplant patients and evaluate the effect of covariates to address variability.
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