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Two classes of receptors involved in cholinergic transmission have been suggested as drug targets for AD: nicotinic ion channels, which mediate fast postsynaptic transmission and muscarinic G protein-coupled ACh receptors.

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Improved” or “stable” galantamine-treated patients displayed mean improvement in ADAS-Cog 11 scores over baseline until 18 months after starting treatment, with lessened deterioration thereafter.

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Xray diffraction data for the study were collected at beamline 19ID of the Argonne Photon Source, which is supported by the Department of Energy and the National Center for Research Resources and National Institute of General Medical Sciences at the NIH.

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For comparison with results obtained with galantamine, the inhibition produced by carbachol was also determined.

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From previous studies (see above), it has been determined that 1 μ m galantamine saturates the binding sites involved in activation by galantamine, whereas 1 μ m galantamine produces little channel block.

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